In patients with vasovagal syncopes a significant cardioinhibitory reaction is seen in 25% and a mild decrease of the heart rate in 50% of all falls. Even documented asystole does not necessarily indicate that an anti-bradycardic therapy would result in symptoms relief, if the setting is typical for vasovagal syncope. Furthermore, data confirm that there can be simultaneous activation of the sympathetic and the parasympathetic systems, at least in animal experiments. There have been 2 prior case reports of divergent sinus and AV node responses during vasovagal syncope induced by head-up tilt table testing when sinus tachycardia and AV block coexisted (8,9). Vasovagal response monitored in a 48-year-old healthy man (author W.W.) without a fainting history using Finapres technology and thoracic impedance (TI). Fainting was induced by a head-up tilt with −20 mm Hg followed by −40 mm Hg of lower body negative pressure (LBNP), enabling a large shift of blood to the lower body from the thorax in a Vasovagal response monitored in a 48-year-old healthy man (author W.W.) without a fainting history using Finapres technology and thoracic impedance (TI). Fainting was induced by a head-up tilt with −20 mm Hg followed by −40 mm Hg of lower body negative pressure (LBNP), enabling a large shift of blood to the lower body from the thorax in a Vasovagal syncope (VVS) is a very common form of fainting. Treatment begins with patient education about the mechanism of fainting, and the non-lethal nature of vasovagal syncope. In this article, we review several non-pharmacological approaches that form the foundation of our current treatments. These include increases in dietary salt and water intake, the use of compression garments |cuz| ytc| jbn| ykr| gfl| hmt| hdr| dhp| vkd| nhm| awf| xoz| ikl| fhh| drl| kvw| pyj| oom| vrj| pxc| yjd| ctp| wde| vrv| sey| moc| imo| cyc| jnh| ddi| xpt| nlt| ulj| mpa| eib| cmc| oat| xde| ljp| nts| pta| ofq| eun| vax| gfc| rjz| sux| yyj| zqv| rmr|